The sense of touch is mediated by specialized receptors located throughout the skin and other tissues, including mechanoreceptors, thermoreceptors, and nociceptors. These receptors detect tactile sensations, pressure, temperature, and pain transmitting signals to the brain for interpretation and response.
Insulin resistance, hyperinsulinemia, and metabolic syndrome can impact peripheral nerves and vascular function, potentially contributing to neuropathic pain, sensory disturbances, and impaired wound healing. Individuals with diabetes are particularly susceptibl to peripheral neuropathy, which can affect tactile sensation and increase the risk of foot ulcers and injuries.
Obesity-related conditions like diabetic neuropathy can lead to alterations in somatosensation, including tingling, numbness, and pain in the extremities. Additionally, excess weight can place pressure on nerves and blood vessels, further compromising sensory function.
Diabetic neuropathy commonly affects somatosensation, leading to sensations of tingling, burning, or numbness, particularly in the hands and feet. Changes in somatosensation can impair balance, coordination, and the ability to detect injuries or wounds.
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